Prenatal hypoxia is a condition characterized by a decrease in uterine oxygen level and can be attributed to 23% of newborn mortality. It can lead to a range of outcomes, from normal development to severe neurological impairments. To further understand this variability, I build upon the work of the Cristancho Lab, understanding the correlation between the epigenome and the metabolome. I used metabolomic approaches to test the role of lactate and protein lactylation as a biomarker of hypoxic injury. Understanding the metabolic consequences of hypoxia will further our understanding of the intricate mechanisms behind neurological damage and recovery.